Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

学研究显示有严重高磷症、低钙症和代谢性中毒。

Amino acids and other organic acids – contribute to acidosis, aciduria, and dysrhythmia.

氨基和其他有机—中毒，尿和节律障碍。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性中毒，就可以认为是继发病。

Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

血液学研究显示有严重高磷血症、低钙血症和代谢性中。

Amino acids and other organic acids – contribute to acidosis, aciduria, and dysrhythmia.

和其他有机—中，尿和节律障碍。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性中，就可以认为是继发病。

Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

血液学研究显示有严重高磷血症、低钙血症性中毒。

Amino acids and other organic acids – contribute to acidosis, aciduria, and dysrhythmia.

氨基其他有机—中毒，尿节律障碍。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性中毒，就可以认为是继发病。

Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

液学研究显示有严重高磷、低钙和谢性中毒。

Amino acids and other organic acids – contribute to acidosis, aciduria, and dysrhythmia.

氨基和其他有机—中毒，尿和节律障碍。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性中毒，就可以认为是继发病。

Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

血学研究显示有严重高磷血症、低钙血症和代谢性中毒。

Amino acids and other organic acids – contribute to acidosis, aciduria, and dysrhythmia.

氨基和其他有—中毒，尿和节律障碍。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性中毒，就可以认为是继发病。

Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

血液学研究显示有严重高磷血症、低钙血症和代谢性中毒。

Amino acids and other organic acids – contribute to acidosis, aciduria, and dysrhythmia.

氨基和其他有机—中毒，尿和节律障碍。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性中毒，就可以认为是继发病。

Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

液学研究显示有严重高磷症、症和代谢性中毒。

Amino acids and other organic acids – contribute to acidosis, aciduria, and dysrhythmia.

氨基和其他有机—中毒，尿和节律障碍。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高尿如伴有家族性肾小管性中毒，就可以认为是继发病。

Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

液学研究显示有严重高磷、低钙和代谢性中毒。

Amino acids and other organic acids – contribute to acidosis, aciduria, and dysrhythmia.

氨基和其他有机—中毒，尿和节律障碍。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性中毒，就可以认为是继发病。

Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

液学研究显示有严重高磷症、低钙症和代谢性中毒。

Amino acids and other organic acids – contribute to acidosis, aciduria, and dysrhythmia.

氨基和其他有机—中毒，尿和节律障碍。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性中毒，就可以认为是继发病。